陳建同
陳建同（Chen Jiann-Thorng）（1962年－）是中華民國陸軍中將，生於台灣省花蓮縣花蓮市，現任國防部軍醫局中將局長，曾任國防部軍醫局少將副局長、國防醫學院上校教學副院長、醫學系主任、陸軍後勤指揮部軍醫處少將處長，畢業於花崗國中、花蓮高中、國防醫學院醫學 系、獲得美國紐約州立大學哲學博士。三軍總醫院眼科部主治醫師。 中華民國眼科醫學會理事長 。學術專長：分子生物學，細胞學，病理學，眼科。
1998年，陳建同以論文: The Nature of the resistance to steroid hypertension in Wistar-Furth rats. 獲得紐約州立大學水牛城分校博士學位；指導教授是Dr.Samuel Gallant博士。
目录
生平
醫療糾紛
博士論文爭議
陳建同博士所發表的論文，與Dr. Yu, Cheng-Ping之相似度高達百分之60，有學術剽竊plagiarism之可能性。由於近年來國軍高階將官論文抄襲事件層出不窮，前國防部長楊念祖閃辭，震驚全國，也啟動教育部對於高階將官博士論文之審查。 陳建同博士論文。 但反方辯護，陳建同博士1998年發表的博士論文，只有在結構和實驗設計上與Dr. Yu 於1997年所發表的博士論文，有部分雷同之處，並無學術剽竊的事實。因為在撰寫論文時，結構體上過於相似，造成誤解。雙方論點，仍然需協力廠商學術單位公證。因此，有關論文爭議事件，完全屬於假設，並非事實。
陳建同博士於1998年，發表的博士論文，題目如下:
- "The Nature of the Resistance to Steroid Hypertension in Wistar Furth Rats"
於1997年，Dr. Yu 所發表的博士論文，題目如下:
- "The Nature of the Resistance to Steroid Hypertension in Wistar Furth Rats"
- "Studies of Steroid-induced Hypertension in Wistar and Wistar/Furth Rats"
- 經過文本分析，內容相似度頗高，且實驗方法與設計，幾乎相同；英文的用字也非常雷同；由於發表的年分接近，學術剽竊的疑慮仍然存在，目前尚待查正並釐清。
論文摘要分析
協力廠商，對於兩位學者的論文摘要進行比對分析，結果如下。
Dr. Yu, 1997, Studies of Steroid-induced Hypertension in Wistar and Wistar/Furth Rats 論文摘要:
陳建同博士，1998年，The Nature of the Resistance to Steroid Hypertension in Wistar Furth Rats論文摘要:
“
Abstract
The Nature of the resistance of the Wistar-Furth(W/fu) rat strain to steroid hypertension was investigated.The basal levels of renal kallikrein mRNA and kallikrein activity measured in intact or uninephrectomized animals were higher in W/Fu than in WI rats. The induction of renal kallikrein mRNA in response to DOCA treatment was similar in both strains. However Renal kallikrein activity increased progressively in W/Fu rats and declined in WI rats. Both urinary total and active kallikrein activities were higher in DOCA-treated W/Fu rats than in the similar treated WI rats.
Co-adminstration of DOCA and Hoe 140, a bradykinin type 2 receptor antagonist, raised the blood pressure of W/Fu rats into the hypertension range. Furthermore, with this regimen W/Fu rats developed hypernatremia. These findings suggest that bradykinin(a product of kallikrein action) plays an important role in the resistance of W/Fu rats to mineralocorticoid hypertension. Collectively, these results suggest that W/Fu rats have a more active renal kallikrein-kinin system.
”
The Nature of the resistance of the Wistar-Furth(W/fu) rat strain to steroid hypertension was investigated.The basal levels of renal kallikrein mRNA and kallikrein activity measured in intact or uninephrectomized animals were higher in W/Fu than in WI rats. The induction of renal kallikrein mRNA in response to DOCA treatment was similar in both strains. However Renal kallikrein activity increased progressively in W/Fu rats and declined in WI rats. Both urinary total and active kallikrein activities were higher in DOCA-treated W/Fu rats than in the similar treated WI rats.
Co-adminstration of DOCA and Hoe 140, a bradykinin type 2 receptor antagonist, raised the blood pressure of W/Fu rats into the hypertension range. Furthermore, with this regimen W/Fu rats developed hypernatremia. These findings suggest that bradykinin(a product of kallikrein action) plays an important role in the resistance of W/Fu rats to mineralocorticoid hypertension. Collectively, these results suggest that W/Fu rats have a more active renal kallikrein-kinin system.
辯護方認為，雖然實驗設計方法類似，但實驗結果有差異，這本屬醫學研究上可容許範圍，雖然結構文體上有相似度，仍然不構成學術剽竊Plagiarism之定義上事實。因此，論文剽竊相關事件不存在。