陈建同
陈建同（Chen Jiann-Thorng）（1962年－）是中华民国陆军中将，生于台湾省花莲县花莲市，现任国防部军医局中将局长，曾任国防部军医局少将副局长、国防医学院上校教学副院长、医学系主任、陆军后勤指挥部军医处少将处长，毕业于花岗国中、花莲高中、国防医学院医学 系、获得美国纽约州立大学哲学博士。三军总医院眼科部主治医师。 中华民国眼科医学会理事长 。学术专长：分子生物学，细胞学，病理学，眼科。
1998年，陈建同以论文: The Nature of the resistance to steroid hypertension in Wistar-Furth rats. 获得纽约州立大学水牛城分校博士学位；指导教授是Dr.Samuel Gallant博士。
目录
生平
医疗纠纷
博士论文争议
陈建同博士所发表的论文，与Dr. Yu, Cheng-Ping之相似度高达百分之60，有学术剽窃plagiarism之可能性。由于近年来国军高阶将官论文抄袭事件层出不穷，前国防部长杨念祖闪辞，震惊全国，也启动教育部对于高阶将官博士论文之审查。 陈建同博士论文。 但反方辩护，陈建同博士1998年发表的博士论文，只有在结构和实验设计上与Dr. Yu 于1997年所发表的博士论文，有部分雷同之处，并无学术剽窃的事实。因为在撰写论文时，结构体上过于相似，造成误解。双方论点，仍然需协力厂商学术单位公证。因此，有关论文争议事件，完全属于假设，并非事实。
陈建同博士于1998年，发表的博士论文，题目如下:
- "The Nature of the Resistance to Steroid Hypertension in Wistar Furth Rats"
于1997年，Dr. Yu 所发表的博士论文，题目如下:
- "The Nature of the Resistance to Steroid Hypertension in Wistar Furth Rats"
- "Studies of Steroid-induced Hypertension in Wistar and Wistar/Furth Rats"
- 经过文本分析，内容相似度颇高，且实验方法与设计，几乎相同；英文的用字也非常雷同；由于发表的年分接近，学术剽窃的疑虑仍然存在，目前尚待查正并厘清。
论文摘要分析
协力厂商，对于两位学者的论文摘要进行比对分析，结果如下。
Dr. Yu, 1997, Studies of Steroid-induced Hypertension in Wistar and Wistar/Furth Rats 论文摘要:
陈建同博士，1998年，The Nature of the Resistance to Steroid Hypertension in Wistar Furth Rats论文摘要:
“
Abstract
The Nature of the resistance of the Wistar-Furth(W/fu) rat strain to steroid hypertension was investigated.The basal levels of renal kallikrein mRNA and kallikrein activity measured in intact or uninephrectomized animals were higher in W/Fu than in WI rats. The induction of renal kallikrein mRNA in response to DOCA treatment was similar in both strains. However Renal kallikrein activity increased progressively in W/Fu rats and declined in WI rats. Both urinary total and active kallikrein activities were higher in DOCA-treated W/Fu rats than in the similar treated WI rats.
Co-adminstration of DOCA and Hoe 140, a bradykinin type 2 receptor antagonist, raised the blood pressure of W/Fu rats into the hypertension range. Furthermore, with this regimen W/Fu rats developed hypernatremia. These findings suggest that bradykinin(a product of kallikrein action) plays an important role in the resistance of W/Fu rats to mineralocorticoid hypertension. Collectively, these results suggest that W/Fu rats have a more active renal kallikrein-kinin system.
”
The Nature of the resistance of the Wistar-Furth(W/fu) rat strain to steroid hypertension was investigated.The basal levels of renal kallikrein mRNA and kallikrein activity measured in intact or uninephrectomized animals were higher in W/Fu than in WI rats. The induction of renal kallikrein mRNA in response to DOCA treatment was similar in both strains. However Renal kallikrein activity increased progressively in W/Fu rats and declined in WI rats. Both urinary total and active kallikrein activities were higher in DOCA-treated W/Fu rats than in the similar treated WI rats.
Co-adminstration of DOCA and Hoe 140, a bradykinin type 2 receptor antagonist, raised the blood pressure of W/Fu rats into the hypertension range. Furthermore, with this regimen W/Fu rats developed hypernatremia. These findings suggest that bradykinin(a product of kallikrein action) plays an important role in the resistance of W/Fu rats to mineralocorticoid hypertension. Collectively, these results suggest that W/Fu rats have a more active renal kallikrein-kinin system.
辩护方认为，虽然实验设计方法类似，但实验结果有差异，这本属医学研究上可容许范围，虽然结构文体上有相似度，仍然不构成学术剽窃Plagiarism之定义上事实。因此，论文剽窃相关事件不存在。